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A Giant Arteriovenous Malformation of the Abdominal Wall

dc.contributor.authorFigueiredo, Adriana
dc.contributor.authorGueifão, Inês
dc.contributor.authorFidalgo, Helena
dc.contributor.authorTavares, Carolina
dc.contributor.authorAmaral, Carlos
dc.contributor.authorFerreira, Rita
dc.contributor.authorBorges, Nuno
dc.contributor.authorFerreira, Maria Emilia
dc.date.accessioned2025-06-12T09:36:09Z
dc.date.available2025-06-12T09:36:09Z
dc.date.issued2025-06
dc.description.abstractINTRODUCTION: Arteriovenous Malformations (AVMs) are high-fow anomalous connections between the arterial and venous systems composed of dysplastic vessels resulting from aberrant angiogenesis. They are congenital and when symptomatic they rarely manifest before adolescence. Depending on the location, size, stage and severity of the symptoms, treatment options vary from conservative management to surgical resection. We report a case of a giant arteriovenous malformation of abdominal wall (tipe IIIb of Yakes Classifcation) treated with surgical resection after prior attempts of scleroembolization.. CLINICAL CASE: 54-year-old woman with known history of osteoarticular pathology and dyspepsia presented a mass on the left side of the abdominal wall with hard consistency, warm, slightly pulsating and tenderness to touch with several years of evolution. The mass showed infltration of the internal and external oblique muscles sparing the transverse muscle. Clinically she presented easy fatigue with efforts. Due to the risk of abdominal wall herniation after excision of the AVM, scleroembolization was considered frst-line treatment in this case. This strategy resulted in regression of the mass and symptoms improvement. Four years after the last intervention, the patient presented lesion growth, recurrence and worsening of symptoms with severe interference in the quality of life (QoL). After multidisciplinary discussion, she was proposed for complete resection of the AVM. She was frst submitted to scleroembolization with Onyx of identifed arterial afferents and sclerosis of the lesion nidus with 2% polidocanol. One month after she underwent successfully total resection of the AVM with the collaboration of General Surgery. CONCLUSION: No unifed agreement exists on the best treatment of these complex high fow lesions and it is diffcult to establish a comprehensive strategy given the pathology’s clinical variability, complex stratifcation and the risk of relapse. A case-by-case approach is needed in managing these types of lesions.eng
dc.identifier.citationAngiol Vasc Surg 2024;20(2):91-93
dc.identifier.doidoi.org/10.48750/acv560
dc.identifier.urihttp://hdl.handle.net/10400.17/5103
dc.language.isoeng
dc.peerreviewedyes
dc.publisherSociedade Portuguesa de Angiologia e Cirurgia Vascular
dc.rights.urihttp://creativecommons.org/licenses/by-nc/4.0/
dc.subjectArteriovenous Malformation
dc.subjectVascular Malformation
dc.subjectPeripheral Arteriovenous Malformation
dc.subjectCongenital Vascular Disease
dc.titleA Giant Arteriovenous Malformation of the Abdominal Walleng
dc.typetext
dspace.entity.typePublication
oaire.citation.endPage93
oaire.citation.issue2
oaire.citation.startPage91
oaire.citation.titleRevista Angiologia e Cirurgia Vascular
oaire.citation.volume20
oaire.versionhttp://purl.org/coar/version/c_970fb48d4fbd8a85

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